
Low Magnesium Impairs Fetal Growth & Promotes Insulin Resistance
Wednesday, November 25, 2009
Byron J. Richards, Board Certified Clinical Nutritionist
Mothers low in magnesium1 are more likely to have a smaller baby, along with low levels of adiponectin Protein hormone that modulates metabolism including glucose and fatty acid catabolism. High levels are associated with low body fat. that set the stage for future insulin resistance and risk for eventually developing type II diabetes. The important issue is that the influences of hormonal signals in the womb and first weeks following birth have a significant programming effect on metabolism that affects a lifetime. In essence, a low birth weight baby is learning a “thrifty metabolism,” meaning that they need to get by with less. This is just fine until such a person starts to overeat later in life, at which point they gain weight far easier than someone who does not have this thrifty setting. We know that magnesium is required for optimal adiponectin Protein hormone that modulates metabolism including glucose and fatty acid catabolism. High levels are associated with low body fat. production. We know that optimal adiponectin Protein hormone that modulates metabolism including glucose and fatty acid catabolism. High levels are associated with low body fat. production is the most important hormone signal for preventing type II diabetes. And this study shows that low magnesium during pregnancy handicaps adiponectin Protein hormone that modulates metabolism including glucose and fatty acid catabolism. High levels are associated with low body fat. production in tandem with low birth weight. Any parent who has had a low birth weight baby should ensure their child learns to like veggies and other good foods while following the Leptin Diet. Once such a child gets off track, the road is likely to be very rocky. Referenced Studies:
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